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In presenza di sintomi di infezione da Herpes virus e della prostata, come la presenza di vesciche in area genitale, o sintomi di meningite herpes virus e della prostata in presenza di fattori di rischio.

Entrambi i tipi sono contagiosi e herpes virus e della prostata in maniera periodica piccole bolle di febbre vesciche che tendono a rompersi e causare delle lesioni. La lesione tende a guarire entro settimane. Le vesciche possono comparire in sede ano-genitale ma anche in corrispondenza dei glutei e delle cosce, e talvolta sono associate a sintomi para-influenzali come febbre e linfonodi ingrossati. Herpes virus e della prostata sintomi di herpes neonatale compaiono durante il primo mese di vita del bambino e, se non trattati, possono causare problemi a lungo termine alla salute del bambino.

Per i casi sospetti di encefalite o meningite, viene prelevato un campione di liquido cefalorachidiano tramite rachicentesi. Per il test sierologico viene prelevato un campione di sangue venoso dal braccio. La ricerca di HSV è utilizzata per rilevare la presenza del virus HSV in pazienti con vesciche o ferite genitali, encefalite ed in neonati nei quali si sospetti herpes neonatale, una condizione rara ma seria, dovuta alla trasmissione del virus dalla madre al nascituro durante il parto.

I metodi di elezione per la ricerca del virus sono rappresentati dalla coltura virale e dalla ricerca del DNA virale PCR. Le forme più gravi di infezione da HSV sono quelle che insorgono in neonati infettati durante il parto o nei soggetti immunocompromessi. In questi le lesioni tendono ad essere più estese e più persistenti che negli individui immunocompetenti.

L'esposizione al virus è continua durante la vita di herpes virus e della prostata i giorni. Salvaggio, M. Herpes Simplex. Medscape Drugs and Diseases. Accessed October Centers for Disease Control and Prevention. Ural, S. Genital Herpes in Pregnancy. Medscape Drugs and Diseases [On-line information]. Devkota, B. Herpes virus e della prostata Simplex Viral Culture.

Sacks, D. Genital herpes. MedlinePlus Medical Encyclopedia [On-line information]. Sharma, R. Herpes Simplex in Emergency Medicine. Couturier, M. Viruses that can lead to cancer. Thomas, Clayton L. Taber's Cyclopedic Medical Dictionary. Pagana, Kathleen D.

Genital Herpes. Wener, K. Herpes genital genital Herpes simplex. AAFP familydoctor. Mayo Clinic Staff. Specialty Laboratories [On-line test information]. Sevall, J. Herpes Simplex Viruses. Herpes: Get the Facts. Herpes More Prevalent Than Thought. Wu, A. Saunders Elsevier, St.

Louis, Missouri. Pp Accessed July Hunt, Richard. Virology: Herpes Viruses Chapter Microbiology and Immunology On-line. University of South Carolina School of Medicine. Lifestyle from Yahoo Canada. Herpes virus - 8 types. Page last reviewed September 6, Accessed January Page last reviewed January 28, American Sexual Health Association.

Fast Facts. Copyright Boston Children's Hospital. Neonatal Herpes Simplex. Last updated Herpes virus e della prostata 21, Mandaci i tuoi commenti.

Noto anche come. Nome ufficiale. Ultima Revisione : Ultima Modifica : In Sintesi. Perché Fare il Test? Quando Fare il Test? Che Tipo di Campione Viene Richiesto? Il Test Richiede una Preparazione?

No, nessuna. Questo test è sensibile e specifico ma richiede due o più giorni. Tramite questa metodica è possibile non solo rilevare il virus ma anche identificarne il tipo. Questo metodo è il migliore per rilevare una meningite, encefalite o cheratite infiammazione della cornea virale da HSV. La concentrazione ematica delle IgG aumenta per alcune settimane per poi diminuire e stabilizzarsi.

Accordion Title. Come viene raccolto il campione per il test? Esiste una preparazione al herpes virus e della prostata che possa assicurare la buona qualità del campione? No, non è necessaria alcuna preparazione. Quali informazioni è possibile ottenere?

Quando viene prescritto? Cosa significa il risultato del test? Pagine Correlate. In questo sito Test. Planned Parenthood: Herpes. Fonti utilizzate nella revisione corrente Salvaggio, M.

Fonti utilizzate nelle precedenti revisioni Thomas, Clayton L. Deborah Fornstrom, M. Kristi Lookner, M.

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Prostate cancer is the second most frequently diagnosed type of cancer and the sixth leading cause of cancer mortality among males worldwide. The aim of this study was to investigate the association between the infection by herpes simplex virus type 2 HSV-2 or herpes virus e della prostata herpesvirus 8 HHV-8 and the risk of prostate cancer. A total of 11 articles with 2, cases and 3, controls were included in herpes virus e della prostata meta-analysis. Further investigations and large-sample studies are required to elucidate the possible mechanism underlying viral carcinogenesis and the association between herpes virus infection and the risk of prostate cancer.

Prostate cancer is the herpes virus e della prostata most frequently diagnosed type of cancer and the sixth leading cause of cancer mortality among males worldwide 1. Prostate cancer is considered a multifactorial disease. Older age, ethnicity and positive family history are well-established risk factors 13.

Genetic factors were considered important in prostate cancer by several studies 4 — 7. Since the s, it has been a matter of debate whether prostate cancer, or at least a subgroup of prostate cancer cases, are associated with infection. With the development of serological assays for the detection of infectious agents, studies were conducted to investigate the relationship between prostate cancer and viral infection 8 — Herpes virus is one of the viruses most commonly related to carcinogenesis.

It was reported that herpes virus plays an important role in the pathogenesis of cancer via the inhibition of cell apoptosis and stimulation of DNA synthesis, which may ultimately lead to cancer Previous studies indicated that infection by herpes simplex virus type 2 HSV-2 or human herpesvirus 8 HHV-8 may be associated with a higher prostate cancer risk 12 — However, other epidemiological studies failed to demonstrate such an association 15 — In order to further assess herpes virus e della prostata correlation between prostate cancer risk and HSV-2 or HHV-8 infection, a meta-analysis was conducted.

Moreover, variant forms of expression were herpes virus e della prostata presence or absence of a hyphen or space between adjacent words, Roman numerals or English words instead herpes virus e della prostata Arabic numerals and presence or absence of quotation marks. Each included publication was reviewed to evaluate whether the following criteria were met: i comparative study assessing the association between infection by HSV-2 or HHV-8 and prostate cancer risk; ii detection method restricted to serological assays; and iii providing sufficient information to determine herpes virus infection incidence number of positive vs.

The inclusion criteria were not restricted by study size, population race or publication language, date or type. For articles with similar population resources or overlapping datasets, only the largest or most recent was included. Studies in which herpes virus was not detected in either group were excluded, since such studies hold no weight in meta-analysis and herpes virus e della prostata not affect conclusions. Two reviewers independently extracted data from all the potentially qualified articles using a standardized data extraction form, in order to avoid mistakes or omissions.

A third reviewer was consulted in the case of discrepancies. Data extracted from the publications included name of the first author, year of publication, number of herpes virus e della prostata and control subjects, exposure frequencies in case and control groups, geographical region and herpes virus subtype.

Subgroup analyses were performed according to geographical region North and South America or Europe. The statistical significance level was set at 0. The extent of heterogeneity among included studies was quantified using the Q test 18 and the I 2 score High I 2 values reflected increasing heterogeneity. A fixed-effects model with the Mantel-Haenszel method was used to provide a summary estimation of the relationship between herpes virus infection and prostate cancer risk when heterogeneity was not significant Otherwise, the random-effects model was utilized We identified a total of 11 articles on herpes virus e della prostata by HSV-2 or HHV-8 and prostate cancer risk, updated up to May 8 — 1114 — 1725 — The process of identifying and selecting articles is shown in Fig.

Four articles were excluded at data extraction, since their results were not considered suitable for this meta-analysis: one of them included a case group only 28one provided ambiguous information regarding the virus subtype 29one lacked a number of negative subjects in the case group and of positive subjects in the control group 30 and one adopted a detection method of inadequate specificity for HSV-2 This meta-analysis consisted of 2, cases and 3, controls.

Studies on different viruses or among separate population groups in the articles mentioned above, were treated as independent studies. Therefore, 19 studies from 11 references were included in this meta-analysis, including 8 on HSV-2 10111415 herpes virus e della prostata, 25 — 27 and 11 on HHV-8 891115 — Detailed characteristics of the eligible studies are listed in Table I. Characteristics of the included studies on herpes simplex virus type 2 HSV-2 or human herpesvirus 8 HHV-8 infection and prostate cancer risk.

Eight studies with 1, cases and 2, controls were included in this meta-analysis, in order to investigate the association between HSV-2 infection and prostate cancer risk.

Five studies were located in North and South America, with 1, cases and 1, controls 11142526 and three were located in Europe, with cases and herpes virus e della prostata 1015 Forest plots of herpes simplex virus type 2 HSV-2 infection and prostate cancer risk for overall analysis. OR, odds ratio; CI, confidence interval. In the stratified analysis by geographical region, no statistically significant heterogeneity was observed in either subgroup Table II.

Therefore, the fixed-effects model was adopted. No publication bias was found in the subgroup analyses, as shown in Table II funnel plot not shown.

Herpes virus e della prostata were 11 studies with 2, cases and 3, controls evaluating the correlation between HHV-8 infection and prostate cancer risk.

There were nine studies in North and South America, with 2, cases and 2, controls 89111617 and two in Europe, with cases and controls herpes virus e della prostata Forest plots of human herpesvirus 8 HHV-8 infection and prostate cancer risk for overall analysis. Stratified analysis was also performed according to geographical region. No association was observed in either subgroup Table II.

There was no publication bias in the overall Fig. We performed sensitivity analyses by sequentially excluding studies one by one, in order to examine the influence of a single study on the overall estimate or on any stratum.

The results demonstrated that no herpes virus e della prostata study considerably affected the summary of risk estimates in this meta-analysis data not shown. Viral infection is a potential risk factor for carcinogenesis. Several studies indicated that these infectious agents may elicit an immune response, creating a cytokine tissue environment that leads to chronic inflammation, DNA damage, cell proliferation, angiogenesis and ultimately prostate cancer 32 — An in vitro study suggested that components of viral and other infectious agents may shift the balance towards altered homeostasis in cells that have already deviated from normal gene expression and may thus play a role in malignant transformation Several epidemiological studies evaluated the association between herpes virus infection and prostate cancer risk, although results were inconsistent 91416 Therefore, an elaborate and comprehensive demonstration of the association between herpes virus infection and prostate cancer risk is of significance.

To the best of our knowledge, this meta-analysis is the first to focus on the relationship between HSV-2 or HHV-8 infection and prostate cancer risk. Prostate cancer incidence and HSV-2 prevalence are affected by geographical region and age. In our meta-analysis, the geographical factor was considered and used for study stratification prior to further analysis. However, no studies from Africa or Asia herpes virus e della prostata included, which restricted a thorough comparison among different areas.

By contrast, older age is a well-established risk factor for prostate cancer 1. The median age of prostate cancer cases in the USA is 67 years, according to the statistics updated up to We aimed to conduct a stratified analysis according to age. However, of all the studies included, only a few provided the mean or median age 101426 Moreover, a study demonstrated that the association with HSV-2 was strengthened when earlier sera analyses were restricted to specimens collected at least five years prior to diagnosis and suggested a long latency period from HSV-2 infection to prostate cancer development We aimed to herpes virus e della prostata this association stratified according to the period between serum sample collection and diagnosis.

However, due to the insufficient information provided, this investigation could not be conducted. Some of the studies reported that the HHV-8 DNA sequence was detected in both normal and cancerous prostatic tissues 39 — 41whereas others did not 42 — Moore and Chang 46 reported that HHV-8 expresses viral interleukin-6, a homolog of human interleukin-6, which was suggested to elicit prostate cancer cell proliferation by Platz and De Marzo Hoffman et al 9 conducted two studies among participants from either the Republic of Trinidad and Tobago or the USA, which demonstrated elevated HHV-8 seropositivity among prostate cancer patients.

The consistent results from two independent laboratories with reliable assays and algorithms were the first documented to indicate the positive association.

However, not all prostate cancer patients in the two studies were HHV-8 seropositive, suggesting that the virus is not always associated with prostate cancer. Another study included in this meta-analysis indicated an inverse association between HHV-8 infection and prostate cancer risk The confounding factor of Mediterranean heritage in the participants enrolled and the differences in assay sensitivity and specificity were considered by the authors as a possible eplanation of the inverse findings, along with the skewing of the immune response mediated by HHV-8 chemokines, which was hypothesized to exert a potentially protective effect on prostate cancer McDonald et al 8 suggested that HHV-8 might segregate patients with manifest and herpes virus e della prostata prostate cancer into two groups, HHV-8 sero-positive prostate cancer detected immediately and HHV-8 sero-negative prostate cancer detected later, which may explain the opposite positive and negative HHV-8 associations observed.

Previous studies detected herpes virus DNA 4147 or anti-virus antibody in tissue samples However, herpes virus detection in tissues was not frequently performed, since blood samples were easier to collect from the participants compared to tissue samples and the serological assays were more convenient to conduct compared to DNA detection. Thus, serological assays were widely used in studies, in order to guarantee an adequate study size and ensure data reliability to a certain extent.

There is no evidence strong enough at present to herpes virus e della prostata that the results of viral infection from blood samples were equal to or comparable with those from tissue samples, therefore, the detection methods for HSV-2 and HHV-8 in this meta-analysis were restricted to serological assays. The pooled ORs were estimated based on all the studies obtained from systematic search, providing a relatively high statistical power.

However, certain limitations should be considered. Firstly, this being a meta-analysis, no original data were obtained. Since prostate cancer is a multifactorial disease and herpes virus infection is related to individual behavior, several covariates, including age, sexual behavior, smoking status and ethnicity, should be taken into consideration to make this meta-analysis more reliable.

However, such information could not be summarized without the original data of the recruited studies, which made it difficult to further assess potential confounding factors. Secondly, although different databases were used to cover herpes virus e della prostata from different areas, there existed a distinction between study quantity and size among different geographical groups, which may bias the conclusions.

No studies from Africa or Asia were included; therefore, assessment regarding these areas was not possible. Furthermore, in HSVrelated analyses, there were five studies with 1, cases and 1, controls in the North and South American group 11142526 and three studies with cases and controls in the European group 1015 Herpes virus e della prostata fewer studies and participants, the results from the European group may not be representative or stable.

Thirdly, the selected databases covered parts of unpublished articles; however, we did not specifically search for these gray data, although no publication bias was observed by tests. In herpes virus e della prostata, the meta-analysis demonstrated a potential association between HSV-2 infection and a higher prostate cancer risk, confined to the North and South American group, as determined by stratified analysis.

An association between HHV-8 infection and prostate cancer was not detected. Further investigations and large-sample studies are required to elucidate herpes virus e della prostata potential mechanism underlying herpes virus e della prostata carcinogenesis and the relationship between herpes virus infection and prostate cancer risk.

National Center for Biotechnology InformationU. Biomed Rep. Published herpes virus e della prostata Mar Author information Article notes Copyright and License information Disclaimer. China, E-mail: nc. Received Dec 20; Accepted Feb

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